Publikované: 16.10.2019

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carb restricted diets work because they lower insulin and so they decrease fat accumulation

http://ajcn.nutrition.org/content/86/4/895.full

https://www.youtube.com/watch?v=Ipvr3dvbh9c

First off, your blood is not a very sweet beverage. Normal fasting blood sugar is slightly less than one teaspoon of sugar in your five or so quarts of blood. What happens when you drink a sugar sweetened Coke that contains 10 teaspoons of sugar?

When you eat any food, even fat, your insulin level will rise. Higher amounts of refined carbohydrates or simple sugars will raise your insulin faster and in higher amounts. The greater the fiber content of your diet, the slower insulin is raised and the more controlled the process.  When you eat a large meal, regardless of the type of calories, it causes a large surge in insulin that is difficult to manage.

Insulin is a taxicab for calories. Its goal is to take blood sugar, as its passenger, to various locations in your body that want it. It helps if you are active, as some of the sugar is more likely to be wanted by cells in your body, including your many muscle cells.

Blood sugar is fuel, like gasoline is to a car. Your brain must have a regular supply or your head conks out. Thus, following a meal your insulin taxi’s are busy transporting sugar through your circulation and out to your cells, hoping to find cells that need some sugar.

In a healthy person, insulin drops off a whopping 60 percent of the sugar at your liver, which acts as a warehouse, converting the blood sugar to glycogen for storage.

Insulin is released by your pancreas in two phases. The first phase is from insulin that is already made and stored in your pancreas, which is just waiting for some food to come along. This is your first wave of taxis coming to pick up the first set of blood sugar passengers. The release of this insulin triggers your pancreas’ beta cells to start making more insulin to deal with the rest of the meal.

As you are eating, some of the insulin transports blood sugar to your white adipose tissue or stored fat. The blood sugar is taken up by fat cells, activating their metabolism, in turn producing the hormone leptin. Leptin now enters your blood and begins traveling up to your brain. The more you eat, the more insulin you make, and the more leptin you make.

When leptin levels get high enough, meaning you have eaten enough, then leptin permeates into your brain and tells your subconscious brain you are full. At the same time, the higher levels of leptin also tell your pancreas that you are full, which turns off the beta cell production of insulin, as no more taxis are needed.

If you ate the right amount of food for your physical activity level, then blood sugar always has some place healthy to go; insulin rises and falls in a controlled manner, as does leptin.

When insulin has too many blood sugar passengers, and cells don’t need any sugar, then insulin stimulates the production of triglycerides, which can become stored fat. This is how you gain weight. Unfortunately, as triglycerides elevate in your blood they get in the way of leptin getting into your brain.  This keeps you eating more than you need to because you don’t yet have a full signal, a problem called leptin resistance.  This encourages even further insulin driven triglyceride formation, making it more likely you will gain weight.

If you stop eating too much and start exercising more, then this simple case issue can improve and will often bounce back to normal function, thus the basic idea of eating less and exercising more to lose weight.

If you continually eat too much and gain weight, then cells get tired of seeing insulin taxis driving up.  In fact, they shutter their windows and lock their doors; insulin becomes persona non grata. The reason for this rejection of insulin is rather simple.  If the cells take in blood sugar when they can’t use it, because they already have enough, then the extra sugar will caramelize and kill the cell.  Rejecting insulin is a self defensive measure. This is the mechanism behind basic insulin resistance at the cellular level.

If this problem goes on, blood sugar levels continue to rise, insulin resistance gets worse, leptin resistance gets worse, cholesterol levels go up, blood pressure goes up, triglyceride levels go up, and inflammation really starts heating up.  Eventually, this leads to type 2 diabetes, along with many risk factors for heart disease, and then heart and kidney disease lock into place.

The problem for any kind of blood sugar medication is that it only addresses one of many mechanisms that aren’t working right, while creating its own side effect complications. At best, it is a temporary solution with a narrow scope of benefit – and does not address the true source of the problem.

Furthermore, when more drugs are added in an effort to more comprehensively address the multiple aspects of the problem, then side effects really pile up in a hurry and injure the patient. This means the risk of multiple drug treatment far outweighs the benefits, even though one or two drugs can’t get the job done.

More often, the medication either forces sugar into cells – killing or injuring them or it transfers the sugar into fat, making leptin problems worse that in turn make insulin problems almost impossible to solve. The shortcoming of these medications is openly acknowledged in the scientific literature2, yet this is what passes for standard medical care.  It is a disgrace.

http://www.wellnessresources.com/tips/articles/insulin_leptin_and_blood_sugar_why_diabetic_medication_fails/

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